Mesenteric traction syndrome during sub-total colectomy

Bhiken Naik MBBCh, Laurie Davies MD
Dept of Anesthesiology, University of Florida

CASE REPORT

A 71-year-old woman, ASA physical status III, with a body mass index of 31.6 was scheduled for subtotal colectomy secondary to persistent rectal bleeding. Her medical history was unremarkable except for long-standing hypertension without clinical evidence of target organ damage. Perioperative laboratory results were normal. On the morning of surgery she was premedicated with midazolam, 2 mg. Anesthesia was induced with thiopental, 5 mg/kg, and fentanyl, 100 ?g; succinylcholine, 100 mg was used for muscle relaxation. After anesthetic induction, the patient was hemodynamically stable with minimal fluctuation in heart rate or blood pressure until a Bookwalter abdominal retractor was secured in position. The patient then developed marked tachycardia with a 50% reduction in systolic blood pressure, and marked flushing of the head and neck area. Retraction was stopped and ephedrine, 20 mg, and phenylephrine, 80 ?g were given in two divided doses without significant improvement in the patient’s condition. Therefore, 10 ?g of epinephrine in two divided doses was given. She responded to the epinephrine bolus doses with both blood pressure and heart rate returning to the pre-traction levels. There was also dramatic resolution of the head and neck flushing. The surgery proceeded uneventfully and the patient was extubated.

DISCUSSION

An association between traction on the abdominal mesentery and a decrease in systolic blood pressure has been documented in patients undergoing abdominal aortic aneurysm repair (1-3). In addition to the hemodynamic changes, a significant number of patients developed marked flushing of the head and neck area (1). The mechanism of the head and neck flushing is not well understood. However, histamine, nucleotides (cyclic AMP + ATP), bradykinins, and prostaglandins have been implicated (1). One study demonstrated that elevated plasma levels of 6-keto-prostaglandin F1 (the stable metabolite of prostacyclin) was significantly associated with decreases in systemic vascular resistance and mean arterial pressure (2). In addition, elevated levels of 6-keto-prostaglandin F1 were associated with marked head and neck flushing. Interestingly, histamine levels remained unchanged with mesenteric traction. Other researchers have demonstrated that ibuprofen pretreatment prevented the mesenteric traction syndrome in patients undergoing abdominal aortic aneurysm repair (3). The strong temporal relationship of the decrease in blood pressure, tachycardia, and head and neck flushing with mesenteric traction in our patient suggest the occurrence of the mesenteric traction syndrome previously described in abdominal aortic aneurysm repair.

References
1. Anesthesiology 1985; 63: 96-99
2. Ann Surgery 1989; 209: 363-367
3. Anesthesiology 1990; 72: 443-44


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