Post Partum Hemorrhage – An Unusual Cause

Kelly Durrett, M.D.
Department of Anesthesiology, University of Florida, Gainesville, Florida

ABSTRACT
A 34-year-old woman, 32 weeks pregnant, presented with preeclampsia. She suffered from a blood pressure of 160/80 mmHg, 2+ proteinuria, and a fetus in breech position. The patient was taken to the operating room for urgent cesarean section. She was given a spinal anesthetic using bupivacaine. Intraoperative vital signs were stable. The obstetrician was unable to extract the fetal head and, therefore, performed a classic (vertical) incision to facilitate delivery. The baby was delivered and the mother continued to have stable vital signs. Following delivery, the mother was given cefazoline (1 g) and oxytocin (40 units). The patient’s vital signs remained stable until the case ended. At this time her systolic blood pressure had decreased to the lower 90s mm Hg. Intravenous (IV) fluids, 2000 ml, were administered for an estimated blood loss of approximately 1000 ml. The abdomen was closed and the patient transported to the postanesthesia care unit (PACU). At that time, the surgeons noticed 500 ml of hemorrhage from the vagina. The uterus felt boggy and uterine atony was diagnosed. Additional oxytocin (40 units) was given IV and carboprost tromethamine (250 mg) was injected intramuscularly..
About one hour after arrival in the PACU, her systolic blood pressure decreased to 40-50 mmHg. The obstetrical service administered additional crystalloid IV fluids and two units packed red blood cells (PRBCs). Her hematocrit was 24%. Her systolic blood pressure continued to be low with signs of shock (cold and clammy skin), crying, and constant pain. The repeat hematocrit was 22% and she was given an additional two units of PRBCs. At this time the anesthesia service was called to place an arterial catheter, but the patient was vasoconstricted, which precluded successful placement. A blood sample was taken to measure arterial blood gas values and demonstrated a hematocrit of 18%. Another two units of PRBCs were administered. In addition, urinary output was substantially decreased. In view of these facts, a central venous catheter was inserted to monitor fluid status and to provide fluid. The patient was then transferred to the SICU.
In the SICU the patient continued to be unstable with low blood pressure, a hematocrit of 24 %, and an INR of 1.7. She was given 1.5 L of 5% albumin, two units PRBCs, and four units of fresh frozen plasma. Abdominal ultrasound was performed and demonstrated a fluid collection at the uterine fundus. Upon paracentesis, the hemoglobin of this fluid was 5 g/dl. No decision was made to return the patient to the operating room. However, the patient remained unstable with systolic blood pressure in the 60s, decreased urinary output, increased shortness of breath, and increased abdominal girth. The patient was returned to the operating room the following afternoon for an exploratory laparotomy, which showed laceration and bleeding of the left ascending uterine artery. The artery was ligated and the patient transported back to the SICU intubated. She was extubated the following day and made a good recovery.

Discussion: This case report illustrates an unusual cause of post partum hemorrhage (PPH), an event that is a potentially life-threatening complication of both vaginal and cesarean delivery. Traditionally, PPH is defined as blood loss greater than 500 ml during vaginal delivery and greater than 1,000 ml during a cesarean delivery. The exact incidence of PPH is difficult to determine, but a reasonable consensus is that 2-4 % of pregnancies are complicated by PPH. More common causes include uterine atony (70%), retained placenta, trauma (uterine rupture, obstetric lacerations), and clotting disorders. Nevertheless, anesthesiologists and obstetricians should maintain vigilance for undiagnosed intra-abdominal trauma such as a lacerated artery when postpartum hemorrhage continues and is recalcitrant to the administration of intravenous fluids and smooth muscle constrictors (i.e., oxytocin, carboprost) of the uterus.
 

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2000 - Cole, Deckinga, Denson, Fuchs, Maples, Naik, Robicsek, R. Zhang

2001 - Denney, Fuchs, Liem, Palacios, Rajasekaran, Rice, Sessions

2002
- Fuchs, Li #1, Li #2, Mayo, Ozcan, Tagalakis,

2003 - Barotti, Barry, Ozcan, Patel, Robinson, Swinney, Tran, van der Heusen , Walters

2004 - Abbasian, Bird, Cahill, Chang, Dahleen, Durret, Horowitz, Perschau, Robinson, Muehlschlegel, Santiago, Velez, Wendling

2005 case reports - Bauernfeind, Cummens, Dagen, Dobija, Yavas

2006 - Book, Chen, Covington, Eisenman, Ficarotta, Hyde, Jordan, Le, Lesko, Moorjani, Muehlschlegel, Seghal, Stine, Tilman